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E.g., 06/03/2026
E.g., 06/03/2026
Justin Perry
About the CNIC
5 Mar 2026

Justin Perry, Associate Member in the Immunology Program of the Sloan Kettering Institute at Memorial Sloan Kettering Cancer Center and an Assistant Professor in the Immunology and Microbial Pathogenesis Program at Weill Cornell Medical Center

From left to right, Borja Ibáñez, Laura Cádiz, Agustín Clemente Moragón, Ángela Pollán, Lucía López Palomar, and Anabel Díaz Guerra.
Research
26 Feb 2026

The study shows that a simple, non-pharmacological technique known as remote ischemic conditioning (RIC) can protect the heart during treatment with anthracyclines

Isabel Fariñas es Profesora de Biología Celular en la Universidad de Valencia (UV) y coordinadora de la unidad de Neurobiología Molecular (NeuroMol)
Raquel Martínez de Meno, Rebeca Acín Pérez, José Antonio Enríquez, Sara Jaroszewicz y Rebeca Sanz-Cortés.
Research
16 Feb 2026

A new CNIC-led study shows that a genetic tool derived from baker’s yeast enables human cells to manufacture the building blocks of DNA even when their mitochondria fail.

call for applications
About the CNIC
3 Feb 2026

The CNIC is opening a public call for entries for visual works, until 15 February, aimed at artists who wish to participate with works in disciplines such as photography, painting, sculpture and mixed media

Equipo del CNIC
Research
2 Feb 2026

A team from the CNIC and Hospital Clínico Universitario de Valladolid has developed a minimally invasive, catheter-based procedure to treat defective mechanical aortic valves

Dra. Ellie Tzima
From left to right: Miguel Sánchez Álvarez, Miguel Ángel del Pozo, David Sancho, Ignacio Heras Murillo, Gillian Dunphy, Aitor Jarit Cabanillas, Elena Moya Ruiz, Irene Adán Barrientos
Research
28 Jan 2026

study published in Immunity reveals that type I interferons (IFN‑I) help resolve inflammation by inducing changes in macrophage mitochondria

Jorge Alegre and Travis Hinson
Research
14 Jan 2026

A CNIC research team has identified for the first time the biological mechanism underlying this susceptibility: high blood pressure creates a hidden metabolic vulnerability in the heart that becomes destabilized when anthracyclines are administered.